Herpesvirus infections of the nervous system
Donald H Gilden, et al.
Nature Clinical Practice Neurology (2007) 3, 82-94
http://www.nature.com/nrneurol/journal/v3/n2/full/ncpneuro0401.html
VZV-associated viremia and lymphotropism have been documented during varicella, herpes zoster, PHN and zoster sine herpete, and even in normal healthy adults.
The detection of VZV DNA [40, 41] and VZV-specific proteins42 in blood MNCs of some patients with PHN and zoster sine herpete, [43, 44, 45] as well as in tissue of patients with chronic VZV ganglionitis, [46] might reflect low-level productive infection in ganglia—a hypothesis that is supported by favorable clinical responses in some PHN patients treated with antiviral agents. [47, 48]
After varicella, the varicella zoster virus (VZV) becomes latent in ganglia along the entire neuraxis; its reactivation can lead to herpes zoster, vasculopathy, myelitis, necrotizing retinitis or zoster sine herpete
see also:
Zoster sine herpete
It would be rash to ignore it
Neurology February 1, 2011 vol. 76 no. 5 416-417
https://www.neurology.org/content/76/5/416.short
6. Varicella-Zoster Virus Infection
Donald H. Gilden
University of Colorado Health Sciences Center
2003. pp 10
ftp://175.45.187.195/ebooks/NEUROLOGI/Clinical%20Neurovirology/DKE97_CH06.pdf
A case of neuropathic brachioradial pruritus caused by cervical disc herniation http://www.nature.com/nrneurol/journal/v4/n6/full/ncpneuro0807.html
Molecular mechanisms of varicella zoster virus pathogenesis
Nature Reviews Microbiology 12, 197–210 (2014)
http://www.nature.com/nrmicro/journal/v12/n3/abs/nrmicro3215.html